Showing posts with label stretching after stroke. Show all posts
Showing posts with label stretching after stroke. Show all posts

Aug 6, 2019

Stretching After Stroke

Address soft tissue shortening.

Until soft tissue shortening is addressed (i.e. muscle tightness), the chronic (post 3 month) survivor has no chance of functional recovery. You can do a ton of hard work but if the muscle length is not there, that's as far as you'll go. It's that simple. This is particularly true of the tendency toward the shortening of soft tissue in the elbow, wrist, finger flexors in the arm and hand. In the leg and foot the main concern is the calf muscle. This muscle often shortens because the calf often has spasticity. Spasticity keeps the foot pointed down in that position, if held long enough will shorten the muscle.

There is a tendency for patients with chronic stroke to limit their stretching of at-risk joints to a few times a day. I would suggest that, given no pathological or orthopedic reasons not to, stretching should be done often. (Always: check with you friendly neighborhood PT or OT!)

Any therapist who works with any patient population with spasticity should know the implications of Botox and intrathecal baclofen, the range of oral medications as well as splinting. The anti-spasticity qualities of these medications are beyond the scope of this article, but they are important in the treatment of spasticity. And therapists are often the clinicians who can redirect patients back to physiatrists and neurologists. These docs then can suggest appropriate meds.

Upon discharge, therapists should "read the riot act" to stroke survivors. Therapists should inform them of the dangers of soft tissue shortening, including decreased function, less chance for future rehabilitation, pain and contracture.

(Note: There is considerable debate about the effectiveness of stretching out spastic muscles. This debate is not among clinicians as much as waged within the world of rehabilitation research. However, even though the scientists are not yet fully convinced, there's reasons to stretch outside of retention of tissue length. For example, the number one cause of poststroke shoulder pain is not subluxation (shoulder separation due to weakness of the shoulder muscles). The number one causes adhesions that build up in the capsule the shoulder. What keeps these adhesions at bay? Stretching. Or at least "ranging." "Ranging" is a term that therapists use to mean not necessary to be stretching, but taking the joint through its full range of motion. Ranging is done passively, as his stretching. That is, stroke survivors limb is moved through its available painless range of motion, but some outside force does it. It might be clinician, a caregiver, or the survivor themselves ranging the joint.)

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Jan 28, 2019

Yes! Stretch!

The small units that make up muscles are called sarcomeres.
Help! Stretch me!
Sarcomeres shorten when we contract our muscles. When we stretch for a long time, there is an increase in the number of sarcomeres. Literally, muscles get longer.


For example, increased flexibility is directly related to an increased number of sarcomeres. One of the ways that the number of sarcomeres can increase happens to all of us: Growth from birth to out 21st year. Growth in the length of bones during childhood provides a prolonged stretch of muscles. As muscles are stretched to their physiological limit they react by developing an increase number of sarcomeres. Stretch has to be of sufficient duration for this remodeling of muscle to occur.

The opposite is true as well. When muscles are left in shortened position, the number of sarcomeres decreases. Nothing provides a prolonged shortening of muscles like spasticity after stroke and brain injury.

So all the rules of stretching are thrown out the window when the muscle is spastic.

How is spastic muscle different than normal muscle? Let me count the ways...

Spastic muscles... 
have lost some (if not all) communication with the brain.
are often kept in a shortened position on the "bad" side for long periods of time.
are not subject to the same rules of stretch. (That rule: The more you stretch the longer the muscle will become.)

Spasticity after brain injury keeps muscles (on the "bad" side) in a shortened position long enough to lose sarcomeres.

It comes as some surprise to most therapists, but there is very little scientific evidence that stretching muscle reduces spasticity. In the very short term there is a small reduction in spasticity. But spasticity is not reduced in any lasting way by stretching because spasticity is not caused by muscles. Spasticity is caused by brain injury. Brain injury causes the brain to cede muscular control to spinal reflexes. Increasing the number of sarcomeres will not reduce spasticity. If it did, every case of spasticity would be eliminated by a regimented stretching program. And wouldn't that be nice?

Soooooo... Should you not stretch? No! I mean yes! I'm confused! Yes, you should stretch!

Why should you stretch? Because even if stretch has no lasting effect on spastic muscle, there are several reasons to do it anyway. Stretch... 
feels good 
reduces spasticity for a sort amount of time 
is good for joints 
may be good for other tissue besides muscle (ligaments, veins/arteries, nerves, skin, etc.)